Cover -- Half-title -- Title -- Copyright -- Contents -- Acknowledgements -- Introduction -- References -- 1 Cognitive function, neuropsychological evaluation, and syndromes of cognitive impairment -- References -- 1.1 Attention -- References -- 1.2 General intelligence, IQ -- References -- 1.3 Memory -- References -- 1.4 Language -- References -- 1.5 Perception -- References -- 1.6 Praxis -- References -- 1.7 Executive function, 'frontal function' -- References -- 1.8 'Bedside' neuropsychological test instruments -- References -- 1.8.1 Mini-Mental State Examination (MMSE) -- References -- 1.8.2 Clock drawing -- References -- 1.8.3 Queen Square Screening Test for Cognitive Deficits -- References -- 1.8.4 Addenbrooke's Cognitive Examination (ACE) and Addenbrooke's Cognitive Examination-Revised (ACE-R) -- References -- 1.8.5 DemTect -- References -- 1.8.6 Dementia Rating Scale (DRS) -- References -- 1.8.7 ADAS-Cog -- References -- 1.8.8 CERAD battery -- References -- 1.8.9 Clinical Dementia Rating (CDR) -- References -- 1.8.10 Global Deterioration Scale (GDS) -- References -- 1.8.11 Instrumental Activities of Daily Living (IADL) scale -- References -- 1.9 Normal aging -- References -- 1.10 Dementia, delirium, depression -- References -- 1.11 Cortical versus subcortical dementias, thalamic dementia -- References -- 1.12 Disconnection syndromes -- References -- Postscript -- References -- 2 Neurodegenerative disorders -- 2.1 Alzheimer's disease (AD) -- Neuropsychological profile -- Attention -- General intelligence, IQ -- Memory -- Language -- Perception -- Praxis -- Executive function -- Presymptomatic Alzheimer's disease -- Treatment of neuropsychological deficits -- References -- 2.2 Frontotemporal lobar degenerations (FTLD) -- References.

2.2.1 Frontotemporal dementia (FTD), dementia of frontal type (DFT), frontal variant of frontotemporal dementia (fvFTD), behavioural variant of frontotemporal dementia (bvFTD) -- Neuropsychological profile -- Attention -- General intelligence, IQ -- Memory -- Language -- Perception -- Praxis -- Executive function -- Treatment of neuropsychological deficits -- References -- 2.2.2 Semantic dementia (SD), progressive fluent aphasia, temporal variant of frontotemporal dementia (tvFTD) -- Neuropsychological profile -- Attention -- General intelligence, IQ -- Memory -- Language -- Perception -- Praxis -- Executive function -- References -- 2.2.3 Progressive non-fluent aphasia (PNFA), primary progressive aphasia (PPA) -- Neuropsychological profile -- Attention -- General intelligence, IQ -- Memory -- Language -- Perception -- Praxis -- Executive function -- References -- 2.2.4 Frontotemporal dementia with parkinsonism linked to chromosome 17 (FTDP-„17) -- References -- 2.2.5 Progressive subcortical gliosis (of Neumann) -- References -- 2.2.6 Argyrophilic grain disease (AGD) -- References -- 2.2.7 Neurofibrillary tangle dementia (NTD), diffuse neurofibrillary tangles with calcification (DNTC, Kosaka-Shibayama disease) -- References -- 2.2.8 Neuronal intermediate filament inclusion disease (NIFID) -- References -- 2.2.9 Basophilic inclusion body disease (BIBD) -- References -- 2.3 Motor neurone disease (MND), amyotrophic lateral sclerosis (ALS) -- Neuropsychological profile -- Attention -- General intelligence, IQ -- Memory -- Language -- Perception -- Praxis -- Executive function -- References -- 2.3.1 Primary lateral sclerosis (PLS), progressive symmetric spinobulbar spasticity -- References -- 2.3.2 Mills' syndrome -- References -- 2.3.3 Hippocampal sclerosis, pure hippocampal sclerosis -- References -- 2.3.4 Progressive muscular atrophy (PMA).

References -- 2.4 Parkinson's disease dementia (PDD) and dementia with Lewy bodies (DLB) -- Neuropsychological profile -- Attention -- General intelligence, IQ -- Memory -- Language -- Perception -- Praxis -- Executive function -- Treatment of neuropsychological deficits -- References -- 2.4.1 Other ('atypical') parkinsonian syndromes -- References -- 2.4.2 Progressive supranuclear palsy (PSP), Steele-Richardson-Olszewski (SRO) syndrome -- REFERENCES -- 2.4.3 Corticobasal degeneration (CBD) -- REFERENCES -- 2.4.4 Multiple system atrophy (MSA) -- References -- 2.4.5 Dementia pugilistica -- References -- 2.4.6 Amyotrophic lateral sclerosis/parkinsonism-dementia complex (ALS/PDC) of Guam, Lytico-Bodig, Marianas dementia -- References -- 2.5 Prion diseases -- References -- 2.5.1 Sporadic prion disease: sporadic Creutzfeldt-Jakob disease (sCJD) -- References -- 2.5.2 Iatrogenic prion disease: variant Creutzfeldt-Jakob disease (vCJD), kuru -- References -- 2.5.3 Inherited prion disease: familial CJD, Gerstmann-Straussler-Scheinker disease (GSS), fatal familial insomnia (FFI) -- References -- 2.6 Mild cognitive impairment (MCI) -- References -- 3 Cerebrovascular disease: vascular dementia and vascular cognitive impairment -- References -- 3.1 Cortical vascular dementia, multi-infarct dementia (MID), post-stroke dementia -- References -- 3.2 Subcortical vascular dementia, Binswanger's disease, lacunar state, subcortical ischaemic vascular disease (SIVD) -- Treatment of neuropsychological deficits in.vascular dementia -- References -- 3.3 Strategic infarct dementia, strategic strokes -- References -- 3.3.1 Angular gyrus -- References -- 3.3.2 Corpus callosum, fornix -- References -- 3.3.3 Thalamus -- References -- 3.3.4 Genu of the internal capsule -- References -- 3.3.5 Caudate nucleus, globus pallidus -- References -- 3.3.6 Hippocampus -- References.

3.3.7 Basal forebrain -- References -- 3.3.8 Brainstem and cerebellum -- References -- 3.4 Subarachnoid haemorrhage (SAH) -- References -- 3.4.1 Aneurysmal SAH, unruptured aneurysms -- References -- 3.4.2 Perimesencephalic (non-aneurysmal) SAH -- References -- 3.4.3 Superficial siderosis of the nervous system -- References -- 3.5 Intracranial vascular malformations -- References -- 3.5.1 Arteriovenous malformations (AVMs) -- References -- 3.5.2 Cavernous haemangiomas -- References -- 3.6 Vasculopathies -- 3.6.1 Angioendotheliomatosis, intravascular’lymphomatosis -- References -- 3.6.2 CADASIL -- References -- 3.6.3 Cerebral amyloid angiopathies (CAA) -- References -- 3.6.4 Familial young-adult-onset arteriosclerotic leukoencephalopathy with alopecia and lumbago without arterial hypertension -- References -- 3.6.5 Familial occipital calcifications, haemorrhagic strokes, leukoencephalopathy, dementia and external carotid dysplasia (FOCHS-LADD) -- References -- 3.6.6 Hereditary endotheliopathy with retinopathy, nephropathy, and stroke (HERNS) -- References -- 3.6.7 Hereditary multi-infarct dementia of’Swedish type -- References -- 3.6.8 Hughes' syndrome (primary antiphospholipid antibody syndrome) -- References -- 3.6.9 Polycythaemia rubra vera -- References -- 3.6.10 Sickle cell disease -- References -- 3.6.11 Sneddon's syndrome -- References -- 3.6.12 Spatz-Lindenberg disease (von Winiwarter-Buerger's disease) -- References -- 3.6.13 Susac syndrome -- References -- 3.7 Other cerebrovascular disorders -- 3.7.1 Cortical venous sinus thrombosis (CVST) -- References -- 3.7.2 Migraine -- References -- 3.7.3 Transient global amnesia (TGA) -- References -- 4 The epilepsies -- 4.1 Epilepsy and cognitive impairment -- References -- 4.2 Cognitive decline and epilepsy: shared aetiology -- 4.2.1 Localization-related (partial) epilepsies -- Temporal lobe epilepsy.

Frontal lobe epilepsy -- References -- 4.2.2 Rasmussen's syndrome (chronic encephalitis and epilepsy) -- References -- 4.2.3 Idiopathic generalized epilepsies -- References -- References -- 4.3 Seizures causing acquired cognitive impairment -- References -- 4.3.1 Transient epileptic amnesia (TEA) -- References -- 4.3.2 Epileptic aphasia, ictal speech arrest -- References -- 4.4 Antiepileptic drug therapy causing cognitive impairment -- References -- 4.5 Treatment of cognitive problems in epilepsy -- References -- 5 Neurogenetic disorders -- 5.1 Hereditary dementias -- 5.1.1 Huntington's disease (HD) -- Neuropsychological profile -- Attention -- Memory -- Language -- Perception -- Praxis -- Executive function -- Presymptomatic gene mutation carriers -- References -- 5.1.2 Dentatorubropallidoluysian atrophy (DRPLA) -- References -- 5.1.3 Familial British dementia (FBD) -- References -- 5.1.4 Familial Danish dementia (FDD) -- References -- 5.1.5 Familial encephalopathy with neuroserpin inclusion bodies (FENIB) -- References -- 5.1.6 Polycystic lipomembranous osteodysplasia with sclerosing leukoencephalopathy (PLOSL), Nasu-Hakola disease, presenile dementia with bone cysts -- References -- 5.1.7 Fahr's syndrome (striatopallidal calcification) -- References -- 5.1.8 Inclusion body myopathy associated with Paget's disease of bone and frontotemporal dementia (IBMPFD) -- References -- 5.1.9 Kufor-Rakeb syndrome (PARK9) -- References -- 5.1.10 Urbach-Wiethe disease (lipoid proteinosis) -- References -- 5.1.11 Fragile X syndrome (FRAX), fragile X tremor/ataxia syndrome (FXTAS) -- References -- 5.2 Hereditary ataxias -- References -- 5.2.1 Autosomal dominant hereditary ataxias, spinocerebellar ataxias (SCA) -- SCA1 -- SCA2 -- SCA3, Machado-Joseph disease (MJD) -- SCA6 -- SCA7 -- SCA8 -- SCA12 -- SCA17 -- SCA19 -- References.

5.2.2 Autosomal recessive hereditary ataxias.