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Alkan, Ayşe Hale, author.
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was silenced or overexpressed in HeLa cells but phenotypic effect couldn’t be detected by apoptosis
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Şengül, Tuğçe, author.
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(apoptosis, ER stress, oxidative stress), altered glycolipid and oligosaccharide metabolism, nerve cell death
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Khan, Nasar, author.
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polyether antibiotics were autophagy inhibitors as well as inducers of apoptosis in cervical, colorectal and
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Gencer, Emel Başak.
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respectively, inhibit apoptosis, induce cell proliferation and develop resistance to chemotherapeutic drugs. In
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Doğan, Hülya, author.
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, expression analysis of p53 targets in APTX knockdown MCF7 cells revealed that extrinsic apoptosis pathway
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Gürer, Dilek Cansu, author.
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cellular processes. To determine which ones are related to cell proliferation and apoptosis in HeLa cells
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Basırlı, Hatice Hande, author.
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myelination, neuron number, and glyco-conjugate content, morphological abnormalities, and apoptosis in brain
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Can, Melike, author.
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cellular processes (ER-oxidative stress, apoptosis), structural abnormalities, glycoconjugate accumulation
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Adan Gökbulut, Aysun, author.
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, apoptosis and cell cycle progression. In this study, genome-wide microarray analysis has been also performed
by
Camgöz, Aylin.
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toxic substances outside of cells. Besides, Bax, which is one of the apoptosis inducing genes, was
by
Efe, Eda, author.
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. Further, the rate of apoptosis was increased in SACM1L overexpressing and knockout MCF10A and MCF7 cells
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Kartal Yandım, Melis, author.
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tumor-suppressor inhibiting cell division, and inducing cell differentiation, senescence and apoptosis. De novo
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