by
Atbinek, Melis, author.
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El Yazması
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– chromatin libraries of HeLa cells. In the future, new libraries can be formed after inducing apoptosis in
by
Şengül, Tuğçe, author.
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(apoptosis, ER stress, oxidative stress), altered glycolipid and oligosaccharide metabolism, nerve cell death
by
Gürler, Sevim Beyza, author.
Format:
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the several pathways including cell proliferation, apoptosis, drug resistance and senescence in cell
by
Olçum Uzan, Melis, author.
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, apoptosis, cell cycle, protein expression, differentiation, cytoskeleton and phenotypic change processes
by
Khan, Nasar, author.
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polyether antibiotics were autophagy inhibitors as well as inducers of apoptosis in cervical, colorectal and
by
Gencer, Emel Başak.
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respectively, inhibit apoptosis, induce cell proliferation and develop resistance to chemotherapeutic drugs. In
by
Yaylak, Bilge, author.
Format:
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AAD + cell death rather than apoptosis when combined with circCLASP1 knockdown. In light of these
by
Doğan, Hülya, author.
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, expression analysis of p53 targets in APTX knockdown MCF7 cells revealed that extrinsic apoptosis pathway
by
Gürer, Dilek Cansu, author.
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cellular processes. To determine which ones are related to cell proliferation and apoptosis in HeLa cells
by
Basırlı, Hatice Hande, author.
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myelination, neuron number, and glyco-conjugate content, morphological abnormalities, and apoptosis in brain
by
Can, Melike, author.
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cellular processes (ER-oxidative stress, apoptosis), structural abnormalities, glycoconjugate accumulation
by
Zeybek Kuyucu, Ayça, author.
Format:
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Alıntı:
. Furthermore, number of tumors was decreased by AKT inhibitor, which was associated with increased apoptosis
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