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Basırlı, Hatice Hande, author.
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altered ganglioside metabolism, ER-stress, oxidative stress, and apoptosis mechanisms in cortex, thalamus
by
Can, Melike, author.
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cellular processes (ER-oxidative stress, apoptosis), structural abnormalities, glycoconjugate accumulation
by
Özdemir, Özgün Öykü, author.
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, they caused serious oxidative stress depending on the protein profile changes related to redox proteins
by
Ateş, Nurselin, author.
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investigated. In the light of our study, impairment in autophagic flux, increased oxidative stress and
by
Cerit, Ender Efe.
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. According to the results, both phenolic acids induced oxidative stress in L. monocytogenes cells. In
18.
by
Erdoğan, Ayşegül, author.
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nitrogen sources, oxidative stress conditions and different light sources on lutein content in green
by
Telli, Kübra, author.
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oxidative stress response markers were analyzed. Furthermore, proteomic analysis was carried out with the
by
Demirtaş, Devran, author.
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1000 ng/ml under oxidative stress conditions. Madecassoside, madecassic acid, asiaticoside, and asiatic
by
Tüncel Çerik, Özge, author.
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of the cells in living systems as mitochondrial disruption, oxidative stress, reactive oxidative
by
Ataç, Beren.
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Alıntı:
cycle control mechanisms and oxidative stress response pathways prompted us to ask whether human p53 was
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