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by
Gürer, Dilek Cansu, author.
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performed with DR5-AS knockdown HeLa cells to uncover which pathways are responsible for these changes. The
by
Sweef, Osama Abdel Hady Biaomy, author.
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processes. We aimed to identify and characterize differentially expressed lncRNAs in apoptosis. HeLa cells
by
Yarımçam, Murat Caner, author.
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was induced in HeLa cells with cisplatin and qRT-PCR was performed with isolated RNAs from the cells
by
Bay, Veysel.
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26 WT. Moreover, in HeLa cells, mutant proteins were observed to be localized mainly in the cytoplasm
by
Kaçar, Vahide İlayda, author.
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morphology of HeLa cells became spherical without inducing apoptosis. Although this lncRNA reduces cell
by
Çiftçi, Yusuf Cem, author.
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apoptosis in HeLa cells. Additionally, when GTF2A1-AS knockdown was combined with cisplatin, it sensitized
by
Çoban, Başak, author.
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models were used to optimize parameters of Mg alginate-based biopatterning method while using HeLa cells
by
Akçaöz, Azime, author.
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fractionation assay showed that translation level decreased in TNF-alpha treated METTL3 KD HeLa cells. As a
by
Efe, Eda, author.
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, hydrolyzes PI4P in the Golgi and ER. Previous studies demonstrated SACM1L depletion in HeLa cells led to
by
Kaya, Meltem, author.
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inhibitory of metastasis of HeLa cells with respect to its effect on cell cycle, migration, p-4EBP1, p-p70S6K
23.
by
Kaya, İbrahim, author.
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. The technique was applied to HeLa (human cervical cancer cells) cells extract for the detection of
by
Aypek, Hande, author.
Format:
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investigated in HeLa, N2A and HaCaT cells. Results suggested that Cx26-I30N and D50Y mutants were not able to
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