by
Begar, Ferit, author.
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cycle life. The cell containing rPOP cathode delivered a discharge capacity of 120 mA h g-1 at a
by
Özel Taşcı, Cansu, author.
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Alıntı:
differences in the cell cycle distributions. As the most significant result, G2 phase distributions were 1.8
by
Yöndem, Eyüp, author.
Format:
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fibroblasts to CAFs is lacking. ECM components may change when cells become cancerous, which can alter cell
by
Başer, Filiz, author.
Format:
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Alıntı:
confirmed with cell cycle analysis and apoptotic cell death. Intestinal glucose absorption was modeled with
by
Kaçar, Vahide İlayda, author.
Format:
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Alıntı:
Cell cycle
by
Efe, Eda, author.
Format:
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Alıntı:
SACM1L in p53 controlled cellular processes like cell cycle and apoptosis in p53 wild type (wt) breast
by
Doğan, Hülya, author.
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processes in p53 wild-type cells through. According to results, Aprataxin overexpression leads to cell cycle
by
Yemeztaşlıca Yetişkin, Egehan, author.
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hours, which is the optimum incubation period, and apoptotic stages and cell cycle stages were compared
by
Erdemli, Kısmet Tuğçe, author.
Format:
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Alıntı:
can dimerize with HIF1-ß to produce HIF that is critical in hypoxic conditions and regulate cell cycle
by
Abdulhadi, Nusaibah, author.
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Alıntı:
Bcl-xL. Besides, the application of DMP caused cell cycle arrest at the G2/M phase. Lastly, DNA damage
by
Gürer, Dilek Cansu, author.
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Alıntı:
Cell cycle.
by
Vural, Ezgi, author.
Format:
El Yazması
Alıntı:
cancer therapy. p53, known as the guardian of genome, controls the cell cycle arrest, apoptosis and DNA
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