12 Results
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by 
Alkan, Ayşe Hale, author.
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El Yazması
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investigated. Candidate lncRNAs from Doxorubicin, Fas mAB, TNF-alpha and Cisplatin treated HeLa cell line were
by 
Kara, Merve, author.
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transcribed from their intronic promoter under cisplatin- and TNF-alpha- mediated apoptosis conditions. In
by 
Yaylak, Bilge, author.
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El Yazması
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/or global circRNAs in apoptotic HeLa cells. Cisplatin (CP), doxorubicin (DOX), Fas mAb(FAS) and
by 
Ahmadov, Ulvi, author.
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%. Under our experimental setting, treatments with cisplatin, doxorubicin, Fas mAb and TNF-alpha led to the
by 
Gürer, Dilek Cansu, author.
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El Yazması
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, a transcriptomics study was performed under cisplatin, doxorubicin, TNF-α and Anti-Fas treatments
by 
Yarımçam, Murat Caner, author.
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El Yazması
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resemblance between GTF2A-1 antisense lncRNA silencing-induced apoptosis and cisplatin-induced apoptosis. The
by 
Demir, Ayşe Banu, author.
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. Among these genes, only PDR5 overexpression was found to have cross-resistance to Cisplatin. Real-time
by 
Çiftçi, Yusuf Cem, author.
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El Yazması
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apoptosis in HeLa cells. Additionally, when GTF2A1-AS knockdown was combined with cisplatin, it sensitized
by 
Telli, Kübra, author.
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El Yazması
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Cisplatin. The clinical success of these inhibitors requires further examination of potential intrinsic or
by 
Kaçar, Vahide İlayda, author.
Format: 
El Yazması
Alıntı: 
novel long non-coding RNA (lncRNA) transcript that is cisplatin inducible and is involved in modulation